The genetic associations identified to date indicate that many different pathways, processes and cell types are involved in generating the SLE phenotype (Fig. 1). Although this interpretation is ...

and showed that MIF regulates a key mediator of inflammatory cell activation. Together with evidence that MIF circulates in increased concentrations in patients with RA and SLE, this information ...

Kinetic analyses revealed that influenza nucleoprotein (NP)-specific BRMs differentiated early after infection and required T follicular helper (Tfh) cell help. BRM differentiation coincided with ...