In atherosclerosis ... followed by a chronic process that provides an essential fibrous cap that prevents plaque rupture. With the recognition of the essential involvement of VSMC proliferation ...

In order to detect a vulnerable atherosclerotic plaque that is more likely to cause cardiovascular events, one should focus on studying biomarkers more associated with destabilization rather than ...

IL-6 secreted by ECs, monocytes, T cells, neutrophils and VSMCs promotes adhesion molecule expression by ECs as well as transmigration of circulating immune cells into the atherosclerotic plaque (this ...

COVID-19 accelerates the progression of atherosclerotic plaques, increasing coronary inflammation and the risk of high-risk ...

They also observed larger necrotic cores, thinner fibrous caps, and greater immune cell content. These findings indicate the development of vulnerable atherosclerotic plaques that are at higher ...

In the 1970s, there was increasing interest in using seaweed-derived polysaccharides as lipid lowering drugs and inhibitors of atherosclerotic plaque formation. However, the discovery of statins ...

This valuable study provides in-vivo evidence that CCR4 regulates the early inflammatory response during atherosclerotic plaque formation. The authors propose that altered T-cell response plays a role ...

In contrast, intraplaque properdin levels were associated with a vulnerable plaque character and symptomatology, potentially reflecting increased deposition of this enhancer of the alternative ...

Adventitial Layer,Aspect Ratio,Atherosclerotic Plaque Rupture,Atherosclerotic Plaques,Calcified Plaques,Carotid Plaque,Cerebrovascular Events,Elevated Stress,Explanation For This Finding,Fiber ...

Atherosclerosis, a key contributor to CVD, is a chronic inflammatory disease that changes the structure of the arterial wall, thereby promoting accumulation of arterial plaque that impairs arterial ...